Transient Neonatal Zinc Deficiency Due to a New Autosomal Dominant Mutation in GeneSLC30A2(ZnT-2)-Reference-Cited by-同舟云学术

Transient Neonatal Zinc Deficiency Due to a New Autosomal Dominant Mutation in GeneSLC30A2(ZnT-2)

Published:2014-01-23 Issue:2 Volume:31 Page:251-252
ISSN:0736-8046
Container-title:Pediatric Dermatology
language:en
Short-container-title:Pediatr Dermatol

Author:

Lova Navarro Miguel¹,Vera Casaño Ángel¹,Benito López Carmen²,Fernández Ballesteros María D.¹,Godoy Díaz Daniel J.¹,Crespo Erchiga Antonio¹,Romero Brufau Santiago³

Affiliation:

1. Department of Dermatology; Hospital Materno-Infantil; Complejo Hospitalario Carlos Haya; Málaga Spain

2. Department of Genetics; Hospital Materno-Infantil; Complejo Hospitalario Carlos Haya; Málaga Spain

3. Mayo Clinic College of Medicine; Rochester Minnesota

Publisher

Wiley

Subject

Dermatology,Pediatrics, Perinatology, and Child Health

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/pde.12257/fullpdf

Reference5 articles.

1. Zinc and skin: a brief summary;Kumar;Dermatol Online J,2012

2. Symptomatic zinc deficiency in a full-term breast-fed infant;Murthy;Dermatol Online J,2010

3. Identification of a mutation in SLC30A2 (ZnT-2) in women with low milk zinc concentration that results in transient neonatal zinc deficiency;Chowanadisai;J Biol Chem,2006

4. A dominant negative heterozygous G87R mutation in the zinc transporter, ZnT-2 (SLC30A2), results in transient neonatal zinc deficiency;Lasry;J Biol Chem,2012

5. Transient zinc deficiency in two full-term breast-fed siblings associated with low maternal breast milk zinc concentration;Glover;Pediatr Dermatol,1988

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