Calmodulin Kinase IV-dependent CREB activation is required for neuroprotection via NMDA receptor-PSD95 disruption-Reference-Cited by-同舟云学术

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Calmodulin Kinase IV-dependent CREB activation is required for neuroprotection via NMDA receptor-PSD95 disruption

Published:2013-03-03 Issue:2 Volume:126 Page:274-287
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Bell Karen F. S.¹,Bent Russell J.¹,Meese-Tamuri Saira¹,Ali Alicia¹,Forder Joan P.¹,Aarts Michelle M.¹

Affiliation:

1. Department of Biological Sciences; University of Toronto; Scarborough Ontario Canada

Funder

Canadian Stroke Network and the Canada Research Chairs Program

Canadian Institutes of Health Research Fellowship

Natural Sciences and Engineering Research Council graduate scholarship

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12176/fullpdf

Reference67 articles.

1. Treatment of ischemic brain damage by perturbing NMDA receptor- PSD-95 protein interactions;Aarts;Science,2002

2. A key role for TRPM7 channels in anoxic neuronal death;Aarts;Cell,2003

3. Pronounced cell death in the absence of NMDA receptors in the developing somatosensory thalamus;Adams;J. Neurosci.,2004

4. CREB activity maintains the survival of cingulate cortical pyramidal neurons in the adult mouse brain;Ao;Mol. Pain,2006

5. Trophic effect of glutamate;Balazs;Curr. Top. Med. Chem.,2006

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