PROTECTION OF ISCHAEMIC-REPERFUSED RAT HEART BY DIMETHYLAMILORIDE IS ASSOCIATED WITH INHIBITION OF MITOCHONDRIAL PERMEABILITY TRANSITION-Reference-Cited by-同舟云学术

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PROTECTION OF ISCHAEMIC-REPERFUSED RAT HEART BY DIMETHYLAMILORIDE IS ASSOCIATED WITH INHIBITION OF MITOCHONDRIAL PERMEABILITY TRANSITION

Published:2007-10-17 Issue:0 Volume:0 Page:071018034236006-???
ISSN:0305-1870
Container-title:Clinical and Experimental Pharmacology and Physiology
language:en
Short-container-title:Clin Exp Pharmacol Physiol

Author:

Prendes María G Marina,Torresín Emilia,González Marcela,Fernández María A,Perazzo Juan C,Savino Enrique A,Varela Alicia

Publisher

Wiley

Subject

Physiology (medical),Pharmacology,Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1440-1681.2007.04800.x/fullpdf

Reference35 articles.

1. The Myocardial Sodium hydrogen Exchanger (NHE) and Its Role in Mediating Ischemic and Reperfusion Injury.

2. Ultrastructural Evidence of the Protective Effect of Na+/H+ Exchange Inhibition on the in vitro Damage Induced by Ischaemia Reperfusion in the Interventricular Septum of the Rabbit Heart

3. Na⁺/H⁺ Exchanger Inhibitor HOE642 Offers Myoprotection in Senescent Myocardium Independent of Ischemic Preconditioning Mechanisms

Cited by 11 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Pharmacological Targeting of the Mitochondrial Permeability Transition Pore for Cardioprotection;Mitochondrial Biology and Experimental Therapeutics;2018

2. Protective Effects of Co-Administration of Gallic Acid and Cyclosporine on Rat Myocardial Morphology Against Ischemia/Reperfusion;Jundishapur Journal of Natural Pharmaceutical Products;2014-10-11

3. Targeting the Mitochondrial Permeability Transition: Cardiac Ischemia-Reperfusion Versus Carcinogenesis;Cellular Physiology and Biochemistry;2011

4. Restricted feeding improves postischemic recovery of Langendorff-perfused rat hearts;Canadian Journal of Physiology and Pharmacology;2010-10

5. A pore way to die: the role of mitochondria in reperfusion injury and cardioprotection;Biochemical Society Transactions;2010-07-26

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