HUMAN HEART ?-ADRENOCEPTORS: ?1-ADRENOCEPTOR DIVERSIFICATION THROUGH ?AFFINITY STATES? AND POLYMORPHISM
Author:
Publisher
Wiley
Subject
Physiology (medical),Pharmacology,Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1440-1681.2007.04730.x/fullpdf
Reference81 articles.
1. 1.AJ Kaumann, L Sanders, and JA Lynhamet al. beta2-Adrenoceptor activation by zinterol causes protein phosphorylation, contractile effects and relaxant effects through a cAMP pathway in human atrium . Mol. Cell. Biochem. 1996 ; 163/164 : 113 -23 .
2. Activation of β 2 -Adrenergic Receptors Hastens Relaxation and Mediates Phosphorylation of Phospholamban, Troponin I, and C-Protein in Ventricular Myocardium From Patients With Terminal Heart Failure
3. Both β 2 - and β 1 -Adrenergic Receptors Mediate Hastened Relaxation and Phosphorylation of Phospholamban and Troponin I in Ventricular Myocardium of Fallot Infants, Consistent With Selective Coupling of β 2 -Adrenergic Receptors to G s -Protein
4. (-)-Adrenaline elicits positive inotropic, lusitropic, and biochemical effects through β2-adrenoceptors in human atrial myocardium from nonfailing and failing hearts, consistent with Gs coupling but not with Gi coupling
5. Intrinsic sympathomimetic activity of (-)-pindolol mediated through a (-)-propranolol-resistant site of the ?1-adrenoceptor in human atrium and recombinant receptors
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