Coexistence of TDP-43 and tau pathology in neurodegeneration with brain iron accumulation type 1 (NBIA-1, formerly Hallervorden-Spatz syndrome)-Reference-Cited by-同舟云学术

Coexistence of TDP-43 and tau pathology in neurodegeneration with brain iron accumulation type 1 (NBIA-1, formerly Hallervorden-Spatz syndrome)

Published:2011-01-30 Issue:5 Volume:31 Page:531-539
ISSN:0919-6544
Container-title:Neuropathology
language:en
Short-container-title:

Author:

Haraguchi Takashi,Terada Seishi,Ishizu Hideki,Yokota Osamu,Yoshida Hidenori,Takeda Naoya,Kishimoto Yuki,Katayama Naoko,Takata Hiroshi,Akagi Motohiro,Kuroda Shigetoshi,Ihara Yuetsu,Uchitomi Yosuke

Publisher

Wiley

Subject

Clinical Neurology,General Medicine,Pathology and Forensic Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1440-1789.2010.01186.x/fullpdf

Reference43 articles.

1. Homozygosity mapping of Hallervorden-Spatz syndrome to chromosome 20p12.3-p13;Taylor;Nat Genet,1996

2. Lewy body in neurodegeneration with brain iron accumulation type 1 is immunoreactive for alpha-synuclein;Arawaka;Neurology,1998

3. A novel pantothenate kinase gene (PANK2) is defective in Hallervorden-Spatz syndrome;Zhou;Nat Genet,2001

4. Pantothenate kinase-associated neurodegeneration (Hallervorden-Spatz syndrome);Gordon;Eur J Paediatr Neurol,2002

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