Abnormal X-Chromosome Dosage Compensation as a Possible Cause of Early Developmental Failure in Mice. (X-chromosome inactivation/trophectoderm/imprinting/embryonic development)
Author:
Publisher
Wiley
Subject
Cell Biology,Developmental Biology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1440-169X.1991.00429.x/fullpdf
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1. The influence of the Robertsonian translocation Rb(X.2)2Ad on anaphase I non-disjunction in male laboratory mice
2. X-chromosome inactivation may explain the difference in viability of XO humans and mice
3. Effects of zero to four copies of chromosome 15 on mouse embryonic development
4. THE NORMAL HUMAN FEMALE AS A MOSAIC OF X-CHROMOSOME ACTIVITY: STUDIES USING THE GENE FOR G-6-PD-DEFICIENCY AS A MARKER
5. A gene from the region of the human X inactivation centre is expressed exclusively from the inactive X chromosome
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1. Misdirection of dosage compensation underlies bidirectional sex-specific death in Wolbachia-infected Ostrinia scapulalis;Insect Biochemistry and Molecular Biology;2015-11
2. The Kinship Theory of Genomic Imprinting;Annual Review of Ecology and Systematics;2000-11
3. Difference in Chromatin Packaging between Active and Inactive X Chromosomes by Fractionation and Allele-Specific Detection;Biochemical and Biophysical Research Communications;1998-03
4. XISTExpression and X-Chromosome Inactivation in Human Preimplantation Embryos;The American Journal of Human Genetics;1997-07
5. Genomic imprinting — defusing the ovarian time bomb;Trends in Genetics;1994-04
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