INVOLVEMENT OF CYTOSKELETAL PROTEINS IN THE MECHANISMS OF ORGANOPHOSPHORUS ESTER-INDUCED DELAYED NEUROTOXICITY
Author:
Publisher
Wiley
Subject
Physiology (medical),Pharmacology,Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1440-1681.1995.tb02015.x/fullpdf
Reference5 articles.
1. The cytoskeleton as a target for organophosphorus ester-induced delayed neurotoxicity (OPIDN)
2. Anomalous phosphorylated neurofilament aggregations in central and peripheral axons of hens treated with tri-Ortho-cresyl phosphate (TOCP)
3. A phosphorylation site in brain and the delayed neurotoxic effect of some organophosphorus compounds
4. Calcium and calmodulin-enhanced in vitro phosphorylation of hen brain cold-stable microtubules and spinal cord neurofilament triplet proteins after a single oral dose of tri-o-cresyl phosphate.
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