An atypicalPMR2  locus is responsible for hypersensitivity to sodium and lithium cations in the laboratory strainSaccharomyces cerevisiae CEN.PK113-7D

Author:

Daran-Lapujade Pascale,Daran Jean-Marc,Luttik Marijke A.H.,Almering Marinka J.H.,Pronk Jack T.,Kötter Peter

Publisher

Oxford University Press (OUP)

Subject

Applied Microbiology and Biotechnology,General Medicine,Microbiology

Reference19 articles.

1. Alepuz PM , Cunninghan KW & Estruch F (1997) Glucose repression affects ion homeostasis in yeast through the regulation of the stress-activated ENA1 gene. Mol Microbiol 26: 91–98.

2. Atack JR , Broughton HB & Pollack SJ (1995) Structure and mechanism of inositol monophosphatase. FEBS Lett 361: 1–7.

3. Bañuelos MA , Sychrová H , Bleykasten-Grosshans C , Souciet JL & Potier S (1998) The Nha1 antiporter of Saccharomyces cerevisiae mediates sodium and potassium efflux. Microbiology 144: 2749–2758.

4. Bro C , Regenberg B , Lagniel G , Labarre J , Montero-Lomelí M & Nielsen J (2003) Transcriptional, proteomic, and metabolic responses to lithium in galactose-grown yeast cells. J Biol Chem 278: 32141–32149.

5. Daran-Lapujade P , Daran JM , Kötter P , Petit T , Piper MDW & Pronk JT (2003) Comparative genotyping of the Saccharomyces cerevisiae laboratory strains S288C and CEN.PK113-7D using oligonucleotide microarrays. FEMS Yeast Res 4: 285–296.

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