Tripchlorolide ameliorates experimental autoimmune encephalomyelitis by down-regulating ERK1/2-NF-κB and JAK/STAT signaling pathways-Reference-Cited by-同舟云学术

Tripchlorolide ameliorates experimental autoimmune encephalomyelitis by down-regulating ERK1/2-NF-κB and JAK/STAT signaling pathways

Published:2015-03-02 Issue:1 Volume:133 Page:104-112
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Zhang Jian¹²,Zeng Yu-qi¹²,Zhang Jing¹²,Pan Xiao-dong¹²,Kang De-yong³,Huang Tian-wen¹²,Chen Xiao-chun¹²

Affiliation:

1. Department of Neurology and Geriatrics; Fujian Institute of Geriatrics; Fujian Medical University Union Hospital; Fuzhou China

2. Key Laboratory of Brain Aging and Neurodegenerative Disease; Fujian Key Laboratory of Molecular Neurology; Fujian Medical University Union Hospital; Fuzhou China

3. Department of Pathology; Fujian Medical University Union Hospital; Fuzhou China

Funder

National Natural Science Foundation of China

Major Project of Fujian Science and Technology Bureau

National and Fujian Province's key Clinical Specialty Discipline Construction Programs of P.R.C

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.13058/fullpdf

Reference47 articles.

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3. The translation of drug efficacy from in vivo models to human disease with special reference to experimental autoimmune encephalomyelitis and multiple sclerosis;Bolton;Inflammopharmacology,2007

4. Activation of NF-kappaB and c-jun transcription factors in multiple sclerosis lesions. Implications for oligodendrocyte pathology;Bonetti;Am. J. Pathol.,1999

5. Corticosteroids in combination therapy for multiple sclerosis;Cohen;Lancet Neurol.,2009

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