Genetic deletion of the prostaglandin E2E prostanoid receptor subtype 3 improves anatomical and functional outcomes after intracerebral hemorrhage-Reference-Cited by-同舟云学术

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Genetic deletion of the prostaglandin E2E prostanoid receptor subtype 3 improves anatomical and functional outcomes after intracerebral hemorrhage

Published:2015-05 Issue:10 Volume:41 Page:1381-1391
ISSN:0953-816X
Container-title:European Journal of Neuroscience
language:en
Short-container-title:Eur J Neurosci

Author:

Leclerc Jenna L.¹²,Lampert Andrew S.¹,Diller Matthew A.¹,Doré Sylvain¹²³

Affiliation:

1. Department of Anesthesiology; University of Florida; 1275 Center Drive Gainesville FL 32610-0159 USA

2. Department of Neuroscience; University of Florida; 1275 Center Drive Gainesville FL 32610-0159 USA

3. Departments of Neurology, Psychiatry and Pharmaceutics; University of Florida; 1275 Center Drive Gainesville FL 32610-0159 USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/ejn.12909/fullpdf

Reference53 articles.

1. Prostaglandin EP1 receptor contributes to excitotoxicity and focal ischemic brain damage;Ahmad;Toxicol. Sci.,2006a

2. 1-HydroxyPGE1 reduces infarction volume in mouse transient cerebral ischemia;Ahmad;Eur. J. Neurosci.,2006b

3. Stimulation of prostaglandin E2-EP3 receptors exacerbates stroke and excitotoxic injury;Ahmad;J. Neuroimmunol.,2007

4. Prostaglandin signalling in cerebral ischaemia;Andreasson;Brit. J. Pharmacol.,2010

5. Molecular pathophysiology of cerebral hemorrhage: secondary brain injury;Aronowski;Stroke,2011

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