FAK contributes to proteinuria in hypercholesterolaemic rats and modulates podocyte F-actin re-organizationviaactivating p38 in response to ox-LDL-Reference-Cited by-同舟云学术

FAK contributes to proteinuria in hypercholesterolaemic rats and modulates podocyte F-actin re-organizationviaactivating p38 in response to ox-LDL

Author:

Hu Mengsi¹,Fan Minghua²,Zhen Junhui³,Lin Jiangong¹,Wang Qun¹,Lv Zhimei¹,Wang Rong¹

Affiliation:

1. Department of Nephrology; Shandong Provincial Hospital Affiliated to Shandong University; Jinan China

2. Department of Obstetrics and Gynecology; The Second Hospital of Shandong University; Jinan China

3. Department of Pathology; School of Medicine; Shandong University; Jinan China

Funder

National Natural Science Foundation of China

Shandong Young Scientists Award Fund

Chinese Society of Nephrology Scientific Fund

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

Reference54 articles.

1. Low density lipoproteins and mitogenic signal transduction processes: role in the pathogenesis of renal disease;Kamanna;Histol Histopathol,2002

5. Early mechanisms of renal injury in hypercholesterolemic or hypertriglyceridemic rats;Joles;J Am Soc Nephrol,2000

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