(Pro)renin receptor (ATP6AP2) depletion arrests As4.1 cells in the G0/G1 phase thereby increasing formation of primary cilia
Author:
Affiliation:
1. Department of Physiology; University Medicine Greifswald; Karlsburg Germany
2. Interfaculty Institute for Genetics and Functional Genomics; University Medicine and Ernst Moritz Arndt-University Greifswald; Greifswald Germany
Funder
Deutsche Forschungsgemeinschaft
Publisher
Wiley
Subject
Cell Biology,Molecular Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/jcmm.13069/fullpdf
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3. The (pro)renin receptor/ATP6AP2 is essential for vacuolar H+-ATPase assembly in murine cardiomyocytes;Kinouchi;Circ Res,2010
4. Prorenin receptor is essential for podocyte autophagy and survival;Riediger;J Am Soc Nephrol,2011
5. A unique exonic splice enhancer mutation in a family with X-linked mental retardation and epilepsy points to a novel role of the renin receptor;Ramser;Hum Mol Genet,2005
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2. Endothelial cell polarity and extracellular matrix composition require functional ATP6AP2 during developmental and pathological angiogenesis;JCI Insight;2022-10-10
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