The autophagic marker p62 highlights Alzheimer type II astrocytes in metabolic/hepatic encephalopathy-Reference-Cited by-同舟云学术

The autophagic marker p62 highlights Alzheimer type II astrocytes in metabolic/hepatic encephalopathy

Published:2020-06-02 Issue:4 Volume:40 Page:358-366
ISSN:0919-6544
Container-title:Neuropathology
language:en
Short-container-title:Neuropathology

Author:

Gelpi Ellen¹²^ORCID,Rahimi Jasmin³,Klotz Sigrid¹,Schmid Susanne¹,Ricken Gerda¹,Forcen‐Vega Sara⁴,Budka Herbert¹,Kovacs Gabor G.¹⁵⁶⁷

Affiliation:

1. Division of Neuropathology and Neurochemistry, Department of Neurology Medical University of Vienna Vienna Austria

2. Neurological Tissue Bank of the Biobank of Hospital Clinic, August Pi i Sunyer Biomedical Research Institute (IDIBAPS) Barcelona Spain

3. Department of Neurology and Karl Landsteiner Institute for Neuroimmunological and Neurodegenerative Disorders Danube Hospital Vienna Austria

4. Neurology Department, Germans Trias i Pujol Hospital Autonomous University of Barcelona Barcelona Spain

5. Center for Neurodegenerative Disease Research (CNDR), Institute on Aging and Department of Pathology and Laboratory Medicine University of Pennsylvania Philadelphia Pennsylvania USA

6. Tanz Centre for Research in Neurodegenerative Disease (CRND) and Department of Laboratory Medicine and Pathobiology University of Toronto Toronto Ontario Canada

7. Laboratory Medicine Program University Health Network Toronto Ontario Canada

Publisher

Wiley

Subject

Clinical Neurology,General Medicine,Pathology and Forensic Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/neup.12660

Reference16 articles.

1. Glial fibrillary acidic protein and S-100 protein in human hepatic encephalopathy: Immunocytochemical demonstration of dissociation of two glia-associated proteins

2. Tubulin polymerization-promoting protein (TPPP/p25) is critical for oligodendrocyte differentiation

3. p62 positive, TDP-43 negative, neuronal cytoplasmic and intranuclear inclusions in the cerebellum and hippocampus define the pathology of C9orf72-linked FTLD and MND/ALS

4. Systematic Screening of Ubiquitin/p62 Aggregates in Cerebellar Cortex Expands the Neuropathological Phenotype of the C9orf72 Expansion Mutation

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