NRF2 addiction in cancer cells-Reference-Cited by-同舟云学术

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NRF2 addiction in cancer cells

Published:2018-03-10 Issue:4 Volume:109 Page:900-911
ISSN:1347-9032
Container-title:Cancer Science
language:en
Short-container-title:Cancer Sci

Author:

Kitamura Hiroshi¹,Motohashi Hozumi¹^ORCID

Affiliation:

1. Department of Gene Expression Regulation Institute of Development, Aging and Cancer Tohoku University Sendai Japan

Funder

Princess Takamatsu Cancer Research Fund

Uehara Memorial Foundation

Mitsubishi Foundation

Japan Society for the Promotion of Science

Naito Foundation

Publisher

Wiley

Subject

Cancer Research,Oncology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cas.13537

Reference80 articles.

1. Nrf2–Keap1 defines a physiologically important stress response mechanism

2. Keap1 Recruits Neh2 through Binding to ETGE and DLG Motifs: Characterization of the Two-Site Molecular Recognition Model

3. Different Electrostatic Potentials Define ETGE and DLG Motifs as Hinge and Latch in Oxidative Stress Response

4. Keap1 is a forked-stem dimer structure with two large spheres enclosing the intervening, double glycine repeat, and C-terminal domains

5. High Sensitivity of Nrf2 Knockout Mice to Acetaminophen Hepatotoxicity Associated with Decreased Expression of ARE-Regulated Drug Metabolizing Enzymes and Antioxidant Genes

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