DNA polymorphisms in linkage disequilibrium at the 3‘ end of the human APO AII gene: relationships with lipids, apolipoproteins and coronary heart disease-Reference-Cited by-同舟云学术

DNA polymorphisms in linkage disequilibrium at the 3‘ end of the human APO AII gene: relationships with lipids, apolipoproteins and coronary heart disease

Published:2008-06-28 Issue:4 Volume:50 Page:191-198
ISSN:0009-9163
Container-title:Clinical Genetics
language:en
Short-container-title:

Author:

Dupuy-Gorce A. M.,Desmarais E.,Vigneron S.,Buresi C.,Nicaud V.,Evans A.,Luc G.,Arveiler D.,Marqués-Vidal P.,Cambien F.,Tiret L.,Paulet A. Crastes,Roizés G.

Publisher

Wiley

Subject

Genetics(clinical),Genetics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1399-0004.1996.tb02624.x/fullpdf

Reference28 articles.

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5. A splice junction mutation responsible for familial apolipoprotein AII deficiency;Deeb;Am J Hum Genet,1990

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1. Apolipoprotein A‐II;High‐Density Lipoproteins;2007-09-20

2. Candidate genes involved in cardiovascular risk factors by a family-based association study on the island of Kosrae, Federated States of Micronesia;American Journal of Medical Genetics;2002-06-11

3. Significant impact of the highly informative (CA)nrepeat polymorphism of theAPOA-IIgene on the plasma APOA-II concentrations and HDL subfractions: The ECTIM study;American Journal of Medical Genetics;2002-04-09

4. Role of apoA-II in lipid metabolism and atherosclerosis: advances in the study of an enigmatic protein;Journal of Lipid Research;2001-11

5. Human Pedigree-Based Quantitative-Trait–Locus Mapping: Localization of Two Genes Influencing HDL-Cholesterol Metabolism;The American Journal of Human Genetics;1999-06