Linkage and segregation analysis of HLA and congenital hypoaldosteronism due to corticosterone methyl-oxydase deficiency type I and type II-Reference-Cited by-同舟云学术

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Linkage and segregation analysis of HLA and congenital hypoaldosteronism due to corticosterone methyl-oxydase deficiency type I and type II

Published:2008-12-11 Issue:1 Volume:24 Page:18-24
ISSN:0001-2815
Container-title:Tissue Antigens
language:en
Short-container-title:

Author:

Jongh B. M. de,Veldhuis J. D.,Drop S. L. S.,Nijenhuis L. E.,Dupont B.

Publisher

Wiley

Subject

Genetics,Biochemistry,Immunology,General Medicine,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1399-0039.1984.tb00392.x/fullpdf

Reference17 articles.

1. No linkage between HLA and congenital hyperplasia due to 11-beta-hydroxylase deficiency;Brautbar;N Engl J Med,1979

2. HLA in a selective aldosterone biosynthetic defect due to type 2 corticosterone methyl-oxidase deficiency;Brautbar;Tissue Antigens,1981

3. Selective hypoaldosteronism in Iranian Jews: an autosomal recessive trait;Cohen;Clin Genet,1977

4. HLA linked genetic control of host response to myco bacterium leprae;Vries;Lancet,1976

5. The effect of ACTH stimulation on plasma steroids in two patients with congenital hypoaldosteronism and their relatives;Drop;Acta Endocrinol,1982

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