Hepatocellular MxA protein expression supports the differentiation of recurrent hepatitis C disease from acute cellular rejection after liver transplantation
Author:
Publisher
Wiley
Subject
Transplantation
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1399-0012.2009.01068.x/fullpdf
Reference28 articles.
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2. Recurrent and acquired hepatitis C viral infection in liver transplant recipients;Wright;Gastroenterology,1992
3. Long-term outcome of hepatitis C infection after liver transplantation;Gane;N Engl J Med,1996
4. Rapidly progressive recurrent hepatitis C virus infection starting 9 days after liver transplantation;Saraf;Liver Transpl,2007
5. Interobserver agreement in hepatitis C grading and staging and in the Banff grading schema for acute cellular rejection: the “hepatitis C 3” multi-institutional trial experience;Netto;Arch Pathol Lab Med,2006
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1. Recurrent Primary Disease After Liver Transplantation;Zakim and Boyer's Hepatology;2018
2. Transplantation Pathology;Macsween's Pathology of the Liver;2018
3. MxA is a positive regulator of type I IFN signaling in HCV infection;Journal of Medical Virology;2017-09-19
4. Lateral flow immunoassay with upconverting nanoparticle‐based detection for indirect measurement of interferon response by the level of MxA;Journal of Medical Virology;2016-09-27
5. Liver transplant complications in hepatitis C infected recipients: recurrence versus rejection;Expert Review of Gastroenterology & Hepatology;2014-03-18
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