Temporal overexpression of IL‐22 and Reg3γ differentially impacts the severity of experimental autoimmune encephalomyelitis-Reference-Cited by-同舟云学术

Temporal overexpression of IL‐22 and Reg3γ differentially impacts the severity of experimental autoimmune encephalomyelitis

Published:2021-05-16 Issue:1 Volume:164 Page:73-89
ISSN:0019-2805
Container-title:Immunology
language:en
Short-container-title:Immunology

Author:

Eken Ahmet¹²^ORCID,Erdem Serife¹²,Haliloglu Yesim¹²,Zehra Okus Fatma¹²,Cakir Mustafa¹²³,Fatih Yetkin Mehmet⁴,Akcakoyunlu Merve⁴,Karayigit Mehmet Onder⁵,Azizoglu Zehra Busra¹²,Bicer Ayten¹²,Gur Tugba Nur¹²,Aslan Kubra¹²,Hora Mehmet²,Oukka Mohamed⁶,Altuntas Hamiyet Donmez¹²,Ufuk Nalbantoglu Ozkan²⁷,Gundogdu Aycan²⁸,Mirza Meral⁴,Canatan Halit¹²

Affiliation:

1. Department of Medical Biology Erciyes University School of Medicine Kayseri Turkey

2. Betül‐Ziya Eren Genome and Stem Cell Center (GENKOK) Kayseri Turkey

3. Department of Medical Biology Van Yuzuncu Yıl University School of Medicine Van Turkey

4. Department of Neurology Erciyes University School of Medicine Kayseri Turkey

5. Department of Pathology, Faculty of Veterinary Medicine Cukurova University Adana Turkey

6. Department of Immunology University of Washington Seattle WA USA

7. Department of Computer Engineering Faculty of Engineering Erciyes University Kayseri Turkey

8. Department of Microbiology and Clinical Microbiology Erciyes University School of Medicine Kayseri Turkey

Funder

Erciyes Üniversitesi

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/imm.13340

Reference66 articles.

1. Border patrol: regulation of immunity, inflammation and tissue homeostasis at barrier surfaces by IL-22

2. IL-22: There Is a Gap in Our Knowledge

3. Microbial Flora Drives Interleukin 22 Production in Intestinal NKp46+ Cells that Provide Innate Mucosal Immune Defense

4. Innate and Adaptive Interleukin-22 Protects Mice from Inflammatory Bowel Disease

5. Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens

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