MAPK15 protects from oxidative stress‐dependent cellular senescence by inducing the mitophagic process-Reference-Cited by-同舟云学术

MAPK15 protects from oxidative stress‐dependent cellular senescence by inducing the mitophagic process

Published:2022-06 Issue:7 Volume:21 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Franci Lorenzo¹²³^ORCID,Tubita Alessandro⁴,Bertolino Franca Maria¹²,Palma Alessandro⁵,Cannino Giuseppe⁶,Settembre Carmine⁷⁸,Rasola Andrea⁶,Rovida Elisabetta⁴,Chiariello Mario¹²^ORCID

Affiliation:

1. Istituto di Fisiologia Clinica (IFC) Consiglio Nazionale delle Ricerche (CNR) Siena Italy

2. Core Research Laboratory (CRL) Istituto per lo Studio la Prevenzione e la Rete Oncologica (ISPRO) Siena Italy

3. Department of Medical Biotechnologies University of Siena Siena Italy

4. Department of Experimental and Clinical Biomedical Sciences University of Firenze Firenze Italy

5. Department of Onco‐hematology, Gene and Cell Therapy Bambino Gesù Children’s Hospital–IRCCS Rome Italy

6. Department of Biomedical Sciences University of Padova Padova Italy

7. Telethon Institute of Genetics and Medicine (TIGEM) Pozzuoli Italy

8. Department of Clinical Medicine and Surgery University of Napoli Federico II Napoli Italy

Funder

Regione Toscana

Associazione Italiana per la Ricerca sul Cancro

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13620

Reference42 articles.

1. Hydroperoxide metabolism in mammalian organs.

2. Cellular Senescence Is Induced by the Environmental Neurotoxin Paraquat and Contributes to Neuropathology Linked to Parkinson’s Disease

3. MAPK15 is part of the ULK complex and controls its activity to regulate early phases of the autophagic process

4. MAPK15 mediates BCR-ABL1-induced autophagy and regulates oncogene-dependent cell proliferation and tumor formation

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