Lymphoid susceptibility to theAggregatibacter actinomycetemcomitanscytolethal distending toxin is dependent upon baseline levels of the signaling lipid, phosphatidylinositol-3,4,5-triphosphate-Reference-Cited by-同舟云学术

Lymphoid susceptibility to theAggregatibacter actinomycetemcomitanscytolethal distending toxin is dependent upon baseline levels of the signaling lipid, phosphatidylinositol-3,4,5-triphosphate

Published:2015-09-24 Issue:1 Volume:31 Page:33-42
ISSN:2041-1006
Container-title:Molecular Oral Microbiology
language:en
Short-container-title:Mol oral Microbiol

Author:

Shenker B.J.¹,Walker L.P.¹,Zekavat A.¹,Boesze-Battaglia K.²

Affiliation:

1. Department of Pathology; University of Pennsylvania School of Dental Medicine; Philadelphia PA USA

2. Department of Biochemistry; University of Pennsylvania School of Dental Medicine; Philadelphia PA USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Microbiology (medical),General Dentistry,Immunology,Microbiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/omi.12127/fullpdf

Reference40 articles.

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2. Cytolethal distending toxin-induced cell cycle arrest of lymphocytes is dependent upon recognition and binding to cholesterol;Boesze-Battaglia;J Biol Chem,2009

3. Regulation of T-cell responses by PTEN;Buckler;Immunol Rev,2008

4. New insights into tumor suppression: PTEN suppresses tumor formation by restraining the phosphoinositide 3 kinase/Akt pathway;Cantley;Proc Natl Acad Sci USA,1999

5. Escherichia coli cytolethal distending toxin blocks the HeLa cell cycle at the G2/M transition by preventing cdc2 protein kinase dephosphorylation and activation;Comayras;Infect Immun,1997

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