Activated KCNQ1 channel promotes fibrogenic response in hereditary gingival fibromatosis via clustering and activation of Ras-Reference-Cited by-同舟云学术

Activated KCNQ1 channel promotes fibrogenic response in hereditary gingival fibromatosis via clustering and activation of Ras

Published:2020-12-31 Issue:3 Volume:56 Page:471-481
ISSN:0022-3484
Container-title:Journal of Periodontal Research
language:en
Short-container-title:J Periodont Res

Author:

Gao Qian¹^ORCID,Yang Chengcan¹,Meng Liuyan¹,Wang Ziming¹,Chen Dong¹,Peng Yao¹,Yang Kai¹,Bian Zhuan¹

Affiliation:

1. The State Key Laboratory Breeding Base of Basic Science of Stomatology (Hubei‐MOST) & Key Laboratory of Oral Biomedicine Ministry of Education School & Hospital of Stomatology Wuhan University Wuhan Hubei China

Funder

Research and Development

Department of Science and Technology, Government of Kerala

Publisher

Wiley

Subject

Periodontics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jre.12836

Reference38 articles.

1. Connective Tissue Metabolism and Gingival Overgrowth

2. Molecular and Clinical Aspects of Drug-induced Gingival Overgrowth

3. Gingival fibromatosis: clinical, molecular and therapeutic issues

4. Antifibrotic Potential of MiR-335-3p in Hereditary Gingival Fibromatosis

5. The KCNQ1 channel - remarkable flexibility in gating allows for functional versatility

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1. Double heterozygous pathogenic mutations in KIF3C and ZNF513 cause hereditary gingival fibromatosis;International Journal of Oral Science;2023-09-26

2. Bioinformatics-based approach to construct a ceRNA network between periodontitis and hereditary gingival fibroplasia;2023-05-12

3. Clinically Relevant KCNQ1 Variants Causing KCNQ1-KCNE2 Gain-of-Function Affect the Ca2+ Sensitivity of the Channel;International Journal of Molecular Sciences;2022-08-26

4. Channelopathy of small- and intermediate-conductance Ca2+-activated K+ channels;Acta Pharmacologica Sinica;2022-06-17