Cap‐independent translation: A shared mechanism for lifespan extension by rapamycin, acarbose, and 17α‐estradiol-Reference-Cited by-同舟云学术

Cap‐independent translation: A shared mechanism for lifespan extension by rapamycin, acarbose, and 17α‐estradiol

Published:2021-03-20 Issue:5 Volume:20 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Shen Ziqian¹,Hinson Abby¹,Miller Richard A.²³^ORCID,Garcia Gonzalo G.²^ORCID

Affiliation:

1. Department of Molecular, Cellular, and Developmental Biology University of Michigan College of Literature, Science, and the Arts Ann Arbor Michigan USA

2. Department of Pathology University of Michigan School of Medicine Ann Arbor Michigan USA

3. University of Michigan Geriatrics Center Ann Arbor Michigan USA

Funder

Glenn Foundation for Medical Research

National Institute on Aging

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13345

Reference41 articles.

1. Sex‐ and tissue‐specific changes in mTOR signaling with age in C57 BL /6J mice

2. Rapamycin blocks the phosphorylation of 4E-BP1 and inhibits cap-dependent initiation of translation.

3. mRNA circularization by METTL3–eIF3h enhances translation and promotes oncogenesis

4. Hyperglycemia Mediates a Shift From Cap-Dependent to Cap-Independent Translation Via a 4E-BP1–Dependent Mechanism

5. Regulation of mTOR Activity in Snell Dwarf and GH Receptor Gene-Disrupted Mice

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