NAD + supplementation prevents STING‐induced senescence in ataxia telangiectasia by improving mitophagy-Reference-Cited by-同舟云学术

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NAD + supplementation prevents STING‐induced senescence in ataxia telangiectasia by improving mitophagy

Published:2021-03-18 Issue:4 Volume:20 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Yang Beimeng¹,Dan Xiuli¹,Hou Yujun¹,Lee Jong‐Hyuk¹,Wechter Noah¹,Krishnamurthy Sudarshan¹,Kimura Risako¹,Babbar Mansi¹,Demarest Tyler¹,McDevitt Ross²,Zhang Shiliang³,Zhang Yongqing⁴,Mattson Mark P.⁵,Croteau Deborah L.¹,Bohr Vilhelm A.¹⁶^ORCID

Affiliation:

1. Laboratory of Molecular Gerontology National Institute on Aging NIH Baltimore MD USA

2. Mouse Phenotyping Unit National Institute on Aging NIH Baltimore MD USA

3. Electron Microscopy Core National Institute on Drug Abuse Intramural Research Program NIH Baltimore MD USA

4. Laboratory of Genetics and Genomics National Institute on Aging National Institutes of Health Baltimore MD USA

5. Department of Neuroscience Johns Hopkins University School of Medicine Baltimore MD USA

6. Danish Center for Healthy Aging University of Copenhagen Copenhagen Denmark

Funder

National Institute on Aging

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13329

Reference43 articles.

1. Interleukin-1β Induces mtDNA Release to Activate Innate Immune Signaling via cGAS-STING

2. Cellular senescence in brain aging and neurodegenerative diseases: evidence and perspectives

3. Nuclear Ataxia-Telangiectasia Mutated (ATM) Mediates the Cellular Response to DNA Double Strand Breaks in Human Neuron-like Cells

4. Review: Activation patterns of microglia and their identification in the human brain

5. CD38 Dictates Age-Related NAD Decline and Mitochondrial Dysfunction through an SIRT3-Dependent Mechanism

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