TL1A/TNFR2‐mediated mitochondrial dysfunction of fibroblast‐like synoviocytes increases inflammatory response in patients with rheumatoid arthritis via reactive oxygen species generation

Author:

Al‐Azab Mahmoud12,Qaed Eskandar3,Ouyang Xunli1,Elkhider Abdalkhalig1,Walana Williams1,Li Han1,Li Weiping1,Tang Yawei1,Adlat Salah4,Wei Jing1,Wang Bing1ORCID,Li Xia1

Affiliation:

1. Department of Immunology College of Basic Medical Science Dalian Medical University Liaoning China

2. Department of Immunology Guangzhou Institute Pediatrics Guangzhou Women and Children Medical Centre Guangzhou Medical University Guangzhou China

3. Department of Pharmacology College of Pharmacy Dalian Medical University Liaoning China

4. Key Laboratory of Molecular Epigenetics of MOE School of Life Science Northeast Normal University Changchun China

Funder

Special Grant for Translational Medicine, Dalian Medical University

The Natural Science Foundation of Liaoning Province of China

College Scientific Research Project of Education Department of Liaoning Province

National Natural Science Foundation of China

Liaoning Distinguished Professor

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

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