Emodin, as a mitochondrial uncoupler, induces strong decreases in adenosine triphosphate (ATP) levels and proliferation of B16F10 cells, owing to their poor glycolytic reserve-Reference-Cited by-同舟云学术

Emodin, as a mitochondrial uncoupler, induces strong decreases in adenosine triphosphate (ATP) levels and proliferation of B16F10 cells, owing to their poor glycolytic reserve

Published:2019-07-28 Issue:8 Volume:24 Page:569-584
ISSN:1356-9597
Container-title:Genes to Cells
language:en
Short-container-title:Genes Cells

Author:

Sugiyama Yuma¹,Shudo Toshiyuki¹,Hosokawa Sho¹,Watanabe Aki¹,Nakano Masaki¹,Kakizuka Akira¹^ORCID

Affiliation:

1. Graduate School of Biostudies, Laboratory of Functional Biology Kyoto University Kyoto Japan

Publisher

Wiley

Subject

Cell Biology,Genetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/gtc.12712

Reference40 articles.

1. Systematic Study of Mitochondrial Toxicity of Environmental Chemicals Using Quantitative High Throughput Screening

2. Uncoupling effect of fungal hydroxyanthraquinones on mitochondrial oxidative phosphorylation

3. Long-Term Metformin Use Is Associated With Decreased Risk of Breast Cancer

4. A hierarchy of ATP-consuming processes in mammalian cells

5. Control of Tumor Bioenergetics and Survival Stress Signaling by Mitochondrial HSP90s

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