Loss of C/EBPδ enhances apoptosis of intestinal epithelial cells and exacerbates experimental colitis in mice-Reference-Cited by-同舟云学术

Loss of C/EBPδ enhances apoptosis of intestinal epithelial cells and exacerbates experimental colitis in mice

Published:2019-07-15 Issue:9 Volume:24 Page:619-626
ISSN:1356-9597
Container-title:Genes to Cells
language:en
Short-container-title:Genes Cells

Author:

Jozawa Hiroki¹,Inoue‐Yamauchi Akane¹^ORCID,Arimura Sumimasa¹,Yamanashi Yuji¹

Affiliation:

1. Division of Genetics, Department of Cancer Biology, The Institute of Medical Science The University of Tokyo Tokyo Japan

Funder

Japan Society for the Promotion of Science

Publisher

Wiley

Subject

Cell Biology,Genetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/gtc.12711

Reference39 articles.

1. C/EBP homologous protein contributes to cytokine-induced pro-inflammatory responses and apoptosis in β-cells

2. The Many Faces of C/EBPδ and their Relevance for Inflammation and Cancer

3. Loss of C/EBPδ enhances IR-induced cell death by promoting oxidative stress and mitochondrial dysfunction

4. Cebpd is essential for gamma‐tocotrienol mediated protection against radiation‐induced hematopoietic and intestinal injury;Banerjee S.;Antioxidants (Basel, Switzerland),2018

5. The Unfolded Protein Response and Chemical Chaperones Reduce Protein Misfolding and Colitis in Mice

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