α2δ‐1 protein drives opioid‐induced conditioned reward and synaptic <scp>NMDA</scp> receptor hyperactivity in the nucleus accumbens-Reference-Cited by-同舟云学术

α2δ‐1 protein drives opioid‐induced conditioned reward and synaptic NMDA receptor hyperactivity in the nucleus accumbens

Published:2022-10-24 Issue:2 Volume:164 Page:143-157
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:Journal of Neurochemistry

Author:

Jin Daozhong¹,Chen Hong¹,Chen Shao‐Rui¹,Pan Hui‐Lin¹^ORCID

Affiliation:

1. Department of Anesthesiology and Perioperative Medicine Center for Neuroscience and Pain Research The University of Texas MD Anderson Cancer Center Houston Texas USA

Funder

National Institute on Drug Abuse

National Institute of Neurological Disorders and Stroke

University of Texas MD Anderson Cancer Center

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jnc.15706

Reference81 articles.

1. Phosphorylation of the N-methyl-d-aspartate receptor is increased in the nucleus accumbens during both acute and extended morphine withdrawal

2. Effect of gabapentin-like compounds on development and maintenance of morphine-induced conditioned place preference

3. Medial Prefrontal Cortex Inversely Regulates Toluene-Induced Changes in Markers of Synaptic Plasticity of Mesolimbic Dopamine Neurons

4. Intraaccumbens administration of NMDA receptor antagonist (±)-CPP prevents locomotor activation conditioned by morphine and amphetamine in rats

5. Locomotor activity in morphine-dependent and post-dependent rats

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