Endogenous elevation of plasma cholecystokinin does not prevent gallstones

Author:

Shahid Rafiq A.1,Wang David Q.-H.2,Fee Brian E.1,McCall Shannon J.3,Romac Joelle M.-J.1,Vigna Steven R.14,Liddle Rodger A.15

Affiliation:

1. Department of Medicine; Duke University Medical Center; Durham NC USA

2. Department of Internal Medicine; Division of Gastroenterology and Hepatology; Saint Louis University School of Medicine; St. Louis MO USA

3. Department of Pathology; Duke University Medical Center; Durham NC USA

4. Department of Cell Biology; Duke University Medical Center; Durham NC USA

5. Durham VA Medical Center; Durham NC USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Clinical Biochemistry,Biochemistry,General Medicine

Reference34 articles.

1. Cholecystokinin bioactivity in human plasma. Molecular forms, responses to feeding, and relationship to gallbladder contraction;Liddle;J Clin Invest,1985

2. Regulation of cholecystokinin secretion by intraluminal releasing factors;Liddle;Am J Physiol,1995

3. Stimulatory effect of an endogenous peptide in rat pancreatic juice on pancreatic enzyme secretion in the presence of atropine: evidence for different mode of action of stimulation from exogenous trypsin inhibitors;Fushiki;Biochem Biophys Res Comm,1984

4. Purification and sequencing of a trypsin-sensitive cholecystokinin-releasing peptide from rat pancreatic juice. Its homology with pancreatic secretory trypsin inhibitor;Iwai;J Biol Chem,1987

5. Feedback regulation by trypsin: evidence for intraluminal CCK-releasing peptide;Miyasaka;Am J Physiol,1989

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