High glucose-induced oxidative stress represses sirtuin deacetylase expression and increases histone acetylation leading to neural tube defects
Author:
Affiliation:
1. Department of Obstetrics, Gynecology & Reproductive Sciences; University of Maryland School of Medicine; Baltimore Maryland USA
2. Department of Biochemistry and Molecular Biology; University of Maryland School of Medicine; Baltimore Maryland USA
Funder
National Institutes of Health
American Diabetes Association
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.13587/fullpdf
Reference59 articles.
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3. Nitrosative stress and redox-cycling agents synergize to cause mitochondrial dysfunction and cell death in endothelial cells;Diers;Redox. Biol.,2013
4. Maternal diabetes triggers DNA damage and DNA damage response in neurulation stage embryos through oxidative stress;Dong;Biochem. Biophys. Res. Commun.,2015
5. MiR-17 down-regulation by high glucose stabilizes Thioredoxin-interacting protein and removes thioredoxin inhibition on ASK1 leading to apoptosis;Dong;Toxicol. Sci.,2016a
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