Effects ofd-Galactosamine Hydrochloride and Partial Hepatectomy on Spontaneous Hepatic Injury and Hepatocarcinogenesis in Long-Evans Cinnamon Rats
Author:
Publisher
Wiley
Subject
Cancer Research,Oncology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1349-7006.1999.tb00775.x/fullpdf
Reference33 articles.
1. Spontaneous hepatic copper accumulation in Long-Evans Cinnamon rats with hereditary hepatitis;Li;J. Clin. Invest.,1991
2. The LEC rat has a deletion in the copper transporting ATPase gene homologous to the Wilson disease gene;Wu;Nat. Genet.,1994
3. New mutation causing hereditary hepatitis in the laboratory rat;Yoshida;J. Hered.,1987
4. High susceptibility to hepatocellular carcinoma development in LEC rats with hereditary hepatitis;Masuda;Jpn. J. Cancer Res.,1988
5. Phenotype of preneoplastic and neoplastic liver lesions during spontaneous liver carcinogenesis of LEC rats;Sawaki;Carcinogenesis,1990
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Prevention of lethal hepatic injury in Long-Evans Cinnamon (LEC) rats by D-galactosamine hydrochloride;The Journal of Medical Investigation;2006
2. Role ofAtp7bGene in Spontaneous andN-Diethylnitrosamine-induced Carcinogenesis in a New Congenic Strain, WKAH.C-Atp7bRats;Japanese Journal of Cancer Research;2001-08
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