Correlation of mutant menin stability with clinical expression of multiple endocrine neoplasia type 1 and its incomplete forms
Author:
Publisher
Wiley
Subject
Cancer Research,Oncology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1349-7006.2011.02055.x/fullpdf
Reference40 articles.
1. Positional cloning of the gene for multiple endocrine neoplasia-type 1;Chandrasekharappa;Science,1997
2. MEN1 gene and its mutations: basic and clinical implications;Tsukada;Cancer Sci,2009
3. Menin, the product of the MEN1 gene, is a nuclear protein;Guru;Proc Natl Acad Sci USA,1998
4. Menin interacts with the AP1 transcription factor JunD and represses JunD-activated transcription;Agarwal;Cell,1999
5. Menin critically links MLL proteins with LEDGF on cancer-associated target genes;Yokoyama;Cancer Cell,2008
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2. Multi-omics analyses of MEN1 missense mutations identify disruption of menin–MLL and menin–JunD interactions as critical requirements for molecular pathogenicity;Epigenetics & Chromatin;2022-08-09
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