Cardiolipin remodeling by ALCAT1 links mitochondrial dysfunction to Parkinson’s diseases-Reference-Cited by-同舟云学术

Cardiolipin remodeling by ALCAT1 links mitochondrial dysfunction to Parkinson’s diseases

Published:2019-03-05 Issue:3 Volume:18 Page:e12941
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Song Chengjie¹²,Zhang Jun³,Qi Shasha⁴,Liu Zhen¹,Zhang Xiaoyang¹,Zheng Yue¹,Andersen John‐Paul²,Zhang Weiping⁴,Strong Randy²,Martinez Paul Anthony²,Musi Nicolas²,Nie Jia²,Shi Yuguang¹²

Affiliation:

1. Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences Nanjing Medical University Nanjing China

2. Barshop Institute for Longevity and Aging Studies, Department of Pharmacology University of Texas Health Science Center San Antonio Texas

3. Perenna Pharmceuticals Inc San Antonio Texas

4. Department of Pathophysiology the Second Military Medical University Shanghai China

Funder

American Diabetes Association

National Institutes of Health

National Natural Science Foundation of China

Nanjing Medical University

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.12941

Reference39 articles.

1. A Novel Cardiolipin-remodeling Pathway Revealed by a Gene Encoding an Endoplasmic Reticulum-associated Acyl-CoA:Lysocardiolipin Acyltransferase (ALCAT1) in Mouse

2. PINK1-Phosphorylated Mitofusin 2 Is a Parkin Receptor for Culling Damaged Mitochondria

3. A Progressive Mouse Model of Parkinson’s Disease: The Thy1-aSyn (“Line 61”) Mice

4. LC3 binds externalized cardiolipin on injured mitochondria to signal mitophagy in neurons

5. Enteric GFAP expression and phosphorylation in Parkinson's disease

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