Cten promotes Epithelial–Mesenchymal Transition (EMT) in colorectal cancer through stabilisation of Src-Reference-Cited by-同舟云学术

Cten promotes Epithelial–Mesenchymal Transition (EMT) in colorectal cancer through stabilisation of Src

Published:2019-07 Issue:7 Volume:69 Page:381-391
ISSN:1320-5463
Container-title:Pathology International
language:en
Short-container-title:Pathol Int

Author:

Asiri Abdulaziz¹²³,Toss Michael S.¹,Raposo Teresa Pereira¹²^ORCID,Akhlaq Maham¹,Thorpe Hannah¹²,Alfahed Abdulaziz¹²⁴,Asiri Abutaleb¹²,Ilyas Mohammad¹²

Affiliation:

1. Division of Cancer and Stem Cells, School of MedicineThe University of Nottingham Nottingham UK

2. Nottingham Molecular Pathology Node, Queen's Medical CentreThe University of Nottingham Nottingham UK

3. Medical Genomics Research Department, King Abdullah International Medical Research Center (KAIMRC), King Saud Bin Abdulaziz University for Health SciencesMinistry of National Guard Health Affairs (MNGH) Riyadh Saudi Arabia

4. Department of Medical Laboratory, College of Applied Medical SciencesPrince Sattam Bin Abdulaziz University Al‐Kharj Saudi Arabia

Publisher

Wiley

Subject

General Medicine,Pathology and Forensic Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/pin.12811

Reference26 articles.

1. Cten, a COOH‐terminal tensin‐like protein with prostate restricted expression, is down‐regulated in prostate cancer;Lo SH;Cancer Res,2002

2. Cten Is Targeted by Kras Signalling to Regulate Cell Motility in the Colon and Pancreas

3. Cten promotes epithelial-mesenchymal transition through the post-transcriptional stabilization of Snail

4. A reciprocal tensin-3–cten switch mediates EGF-driven mammary cell migration

5. Up-regulated cten by FGF2 contributes to FGF2-mediated cell migration

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