Native LDL-induced oxidative stress in human proximal tubular cells: multiple players involved-Reference-Cited by-同舟云学术

Native LDL-induced oxidative stress in human proximal tubular cells: multiple players involved

Published:2011-02 Issue:2 Volume:15 Page:375-395
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:

Author:

Piccoli Claudia,Quarato Giovanni,D’Aprile Annamaria,Montemurno Eustacchio,Scrima Rosella,Ripoli Maria,Gomaraschi Monica,Cirillo Pietro,Boffoli Domenico,Calabresi Laura,Gesualdo Loreto,Capitanio Nazzareno

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1582-4934.2009.00946.x/fullpdf

Reference57 articles.

1. Role of lipid and lipoprotein fractions in atherogenesis: the Framingham Study;Kannel;Prog Lipid Res.,1981

2. Beyond cholesterol. Modifications of low-density lipoprotein that increase its atherogenicity;Steinberg;N Engl J Med.,1989

3. The role of oxidized lipoproteins in atherogenesis;Berliner;Free Radic Biol Med.,1996

4. Oxidized Low-Density Lipoprotein and Oxidative Stress in the Development of Glomerulosclerosis;Lee;Am J Nephrol.,2009

5. Lipoprotein metabolism in chronic renal insufficiency;Saland;Pediatr Nephrol.,2007

Cited by 10 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Research trends of acupuncture therapy for hypertension over the past two decades: a bibliometric analysis;Cardiovascular Diagnosis and Therapy;2023-02

2. Risk Factors of Renal Tubular Dysfunction in Thai People Living with HIV Receiving Tenofovir Disoproxil Fumarate;Journal of the International Association of Providers of AIDS Care (JIAPAC);2022-01

3. Genetic linkage of oxidative stress with cardiometabolic traits in an intercross derived from hyperlipidemic mouse strains;Atherosclerosis;2020-01

4. The Mechanism of Acupuncture in Treating Essential Hypertension: A Narrative Review;International Journal of Hypertension;2019-03-07

5. Para-hydroxyphenylpyruvate inhibits the pro-inflammatory stimulation of macrophage preventing LPS-mediated nitro-oxidative unbalance and immunometabolic shift;PLOS ONE;2017-11-27