Anti-type VII collagen autoantibodies, detected by enzyme-linked immunosorbent assay, fluctuate in parallel with clinical severity in patients with epidermolysis bullosa acquisita
Author:
Affiliation:
1. Department of Dermatology; Keio University School of Medicine; Tokyo Japan
2. Department of Dermatology; Faculty of Medicine; Cairo University; Cairo Egypt
Publisher
Wiley
Subject
Dermatology,General Medicine
Reference18 articles.
1. Identification of the skin basement-membrane auto-antigen in epidermolysis bullosa-acquisita;Woodley;N Engl J Med,1984
2. Epidermolysis bullosa acquisita antigen, a new major component of cutaneous basement membrane, is a glycoprotein with collagenous domains;Woodley;J Invest Dermatol,1986
3. Type VII collagen: the anchoring fibril protein at fault in dystrophic epidermolysis bullosa;Chung;Dermatol Clin,2010
4. The large non-collagenous domain (NC-1) of type VII collagen is amino-terminal and chimeric. Homology to cartilage matrix protein, the type III domains of fibronectin and the A domains of von Willebrand factor;Christiano;Hum Mol Genet,1992
5. The carboxyl-terminal half of type VII collagen, including the non-collagenous NC-2 domain and intron/exon organization of the corresponding region of the COL7A1 gene;Greenspan;Hum Mol Genet,1993
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