Akt acts as a switch for GPCR transactivation of the TGF‐β receptor type 1-Reference-Cited by-同舟云学术

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Akt acts as a switch for GPCR transactivation of the TGF‐β receptor type 1

Published:2021-12-18 Issue: Volume: Page:
ISSN:1742-464X
Container-title:The FEBS Journal
language:en
Short-container-title:The FEBS Journal

Author:

Mohamed Raafat¹,Shajimoon Aravindra¹²,Afroz Rizwana¹,Gabr Mai¹,Thomas Walter G.²,Little Peter J.¹³,Kamato Danielle¹^ORCID

Affiliation:

1. School of Pharmacy Pharmacy Australia Centre of Excellence The University of Queensland Woolloongabba Australia

2. School of Biomedical Sciences Faculty of Medicine The University of Queensland St Lucia Australia

3. Department of Pharmacy Xinhua College of Sun Yat‐sen University Guangzhou China

Funder

National Health and Medical Research Council

National Heart Foundation of Australia

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/febs.16297

Reference60 articles.

1. TGF-β-induced SMAD signaling and gene regulation: consequences for extracellular matrix remodeling and wound healing

2. Mechanisms of TGF-β Signaling from Cell Membrane to the Nucleus

3. TGF‐beta signaling in fibrosis;Biernacka A;Growth Factors,2011

4. Thrombin stimulation of proteoglycan synthesis in vascular smooth muscle is mediated by protease‐activated receptor‐1 transactivation of the transforming growth factor β type I receptor;Burch ML;J Biol Chem,2010

5. Protease activated receptor‐1 mediated dual kinase receptor transactivation stimulates the expression of glycosaminoglycan synthesizing genes;Kamato D;Cell Signal,2016

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