Mutant eIF2B leads to impaired mitochondrial oxidative phosphorylation in vanishing white matter disease-Reference-Cited by-同舟云学术

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Mutant eIF2B leads to impaired mitochondrial oxidative phosphorylation in vanishing white matter disease

Published:2017-04-16 Issue:5 Volume:141 Page:694-707
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Raini Gali¹,Sharet Reut¹,Herrero Melisa¹,Atzmon Andrea¹,Shenoy Anjana²,Geiger Tamar²,Elroy-Stein Orna¹³^ORCID

Affiliation:

1. Department of Cell Research and Immunology; George S. Wise Faculty of Life Sciences; Tel Aviv University; Tel Aviv Israel

2. Department of Human Molecular Genetics and Biochemistry; Sackler Faculty of Medicine; Tel Aviv University; Tel Aviv Israel

3. Sagol School of Neuroscience; Tel Aviv University; Tel Aviv Israel

Funder

The Legacy Heritage Bio-Medical Program of the Israel Science Foundation

Israel Center of Research Excellence program

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.14024/fullpdf

Reference38 articles.

1. Effect of ATP depletion on the palmitoylation of myelin proteolipid protein in young and adult rats;Bizzozero;J. Neurochem.,1999

2. Leukoencephalopathy with vanishing white matter: a review;Bugiani;J. Neuropathol. Exp. Neurol.,2010

3. Defective glial maturation in vanishing white matter disease;Bugiani;J. Neuropathol. Exp. Neurol.,2011

4. Hyaluronan accumulation and arrested oligodendrocyte progenitor maturation in vanishing white matter disease;Bugiani;Brain,2013

5. Poor cerebral inflammatory response in eIF2B knock-in mice: implications for the aetiology of vanishing white matter disease;Cabilly;PLoS ONE,2012

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