Inflammasome activity is essential for one kidney/deoxycorticosterone acetate/salt-induced hypertension in mice-Reference-Cited by-同舟云学术

Inflammasome activity is essential for one kidney/deoxycorticosterone acetate/salt-induced hypertension in mice

Published:2015-07-31 Issue:4 Volume:173 Page:752-765
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:British Journal of Pharmacology

Author:

Krishnan S M¹,Dowling J K²,Ling Y H¹,Diep H¹,Chan C T¹,Ferens D¹,Kett M M³,Pinar A²,Samuel C S¹,Vinh A¹,Arumugam T V⁴⁵,Hewitson T D⁶,Kemp-Harper B K¹,Robertson A A B⁷,Cooper M A⁷,Latz E⁸⁹¹⁰,Mansell A²,Sobey C G¹¹¹,Drummond G R¹¹¹

Affiliation:

1. Department of Pharmacology; Monash University; Clayton Vic. Australia

2. Centre for Innate Immunity and Infectious Diseases; MIMR-PHI Institute of Medical Research; Clayton Vic. Australia

3. Department of Physiology; Monash University; Clayton Vic. Australia

4. Department of Physiology; Yong Loo Lin School of Medicine; National University of Singapore; Singapore

5. School of Pharmacy; Sungkyunkwan University; Suwon Republic of Korea

6. Department of Nephrology; Royal Melbourne Hospital; Parkville Vic. Australia

7. Division of Chemistry and Structural Biology; Institute for Molecular Bioscience; The University of Queensland; Brisbane Australia

8. Institute of Innate Immunity; University Hospital; University of Bonn; Bonn Germany

9. Department of Infectious Diseases and Immunology; University of Massachusetts Medical School; Worcester MA USA

10. German Center for Neurodegenerative Diseases; Bonn Germany

11. Department of Surgery; Monash Medical Centre; Southern Clinical School; Monash University; Clayton Vic. Australia

Funder

National Health and Medical Research Council of Australia (NHMRC)

Group-of-Eight Australia (Go8)/German Academic Exchange Service (DAAD) Joint Research-Cooperation Scheme

Monash Graduate Scholarship

Publisher

Wiley

Subject

Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/bph.13230/fullpdf

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