Expression of CEACAM1 or CEACAM5 in AZ-521 cells restores the type IV secretion deficiency for translocation of CagA byHelicobacter pylori
Author:
Affiliation:
1. Department of Biology, Division of Microbiology; Friedrich Alexander University Erlangen; Erlangen Germany
2. Medical Faculty, Institute of Anatomy; University of Duisburg-Essen; Essen Germany
Funder
Deutsche Forschungsgemeinschaft
Publisher
Hindawi Limited
Subject
Virology,Immunology,Microbiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/cmi.12965/fullpdf
Reference81 articles.
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3. The Helicobacter pylori type IV secretion system encoded by the cag pathogenicity island: Architecture, function, and signaling;Backert;Current Topics in Microbiology and Immunology,2017
4. Subproteomes of soluble and structure-bound Helicobacter pylori proteins analyzed by two-dimensional gel electrophoresis and mass spectrometry;Backert;Proteomics,2005
5. Phosphorylation of tyrosine 972 of the Helicobacter pylori CagA protein is essential for induction of a scattering phenotype in gastric epithelial cells;Backert;Molecular Microbiology,2001
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