Expression of CEACAM1 or CEACAM5 in AZ-521 cells restores the type IV secretion deficiency for translocation of CagA byHelicobacter pylori

Author:

Tegtmeyer Nicole1,Harrer Aileen1,Schmitt Verena2,Singer Bernhard B.2,Backert Steffen1ORCID

Affiliation:

1. Department of Biology, Division of Microbiology; Friedrich Alexander University Erlangen; Erlangen Germany

2. Medical Faculty, Institute of Anatomy; University of Duisburg-Essen; Essen Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Hindawi Limited

Subject

Virology,Immunology,Microbiology

Reference81 articles.

1. Pathobiology of Helicobacter pylori-Induced Gastric Cancer;Amieva;Gastroenterology,2016

2. Emerging roles of Abl family tyrosine kinases in microbial pathogenesis;Backert;Trends Biochemistry Science,2008

3. The Helicobacter pylori type IV secretion system encoded by the cag pathogenicity island: Architecture, function, and signaling;Backert;Current Topics in Microbiology and Immunology,2017

4. Subproteomes of soluble and structure-bound Helicobacter pylori proteins analyzed by two-dimensional gel electrophoresis and mass spectrometry;Backert;Proteomics,2005

5. Phosphorylation of tyrosine 972 of the Helicobacter pylori CagA protein is essential for induction of a scattering phenotype in gastric epithelial cells;Backert;Molecular Microbiology,2001

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