NETs promote ROS production to induce human amniotic epithelial cell apoptosis via ERK1/2 signaling in spontaneous preterm birth-Reference-Cited by-同舟云学术

NETs promote ROS production to induce human amniotic epithelial cell apoptosis via ERK1/2 signaling in spontaneous preterm birth

Published:2022-12 Issue:3 Volume:89 Page:
ISSN:1046-7408
Container-title:American Journal of Reproductive Immunology
language:en
Short-container-title:American J Rep Immunol

Author:

Hu Min¹²^ORCID,Li Hao¹²,Li Guangzhen³,Wang Yuan¹²,Liu Jing¹²,Zhang Meihua²,Shen Di⁴¹²,Wang Xietong⁴¹²

Affiliation:

1. Department of Obstetrics and Gynaecology Shandong Provincial Hospital Shandong University Jinan Shandong China

2. Key Laboratory of Birth Regulation and Control Technology of National Health Commission of China Shandong Provincial Maternal and Child Health Care Hospital Jinan Shandong China

3. Department of General Surgery Qilu Hospital of Shandong University Jinan China

4. Department of Obstetrics and Gynaecology Maternal and Child Health Care Hospital of Shandong Provincial Jinan Shandong China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Obstetrics and Gynecology,Reproductive Medicine,Immunology,Immunology and Allergy,Obstetrics and Gynecology,Immunology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/aji.13656

Reference41 articles.

1. Preterm Birth: Now the Leading Cause of Child Death Worldwide

2. Epidemiology and causes of preterm birth

3. Spontaneous preterm labour that leads to preterm birth: An update and personal reflection

4. Immunologic challenges of human reproduction: an evolving story

5. IL-37 Exerts Anti-Inflammatory Effects in Fetal Membranes of Spontaneous Preterm Birth via the NF-κB and IL-6/STAT3 Signaling Pathway

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