Chronic inhibition of NO synthase enhances the production of prostacyclin in coronary arteries through upregulation of the cyclooxygenase type 1 isoform-Reference-Cited by-同舟云学术

Chronic inhibition of NO synthase enhances the production of prostacyclin in coronary arteries through upregulation of the cyclooxygenase type 1 isoform

Published:1997-05-06 Issue:3 Volume:11 Page:252-259
ISSN:0767-3981
Container-title:Fundamental & Clinical Pharmacology
language:en
Short-container-title:

Author:

Beverelli F.,Béa ML,Puybasset L.,Giudicelli JF,Berdeaux A.

Publisher

Wiley

Subject

Pharmacology (medical),Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1472-8206.1997.tb00193.x/fullpdf

Reference31 articles.

1. Effect of cyclooxygenase blockade on blood flow through well-developed coronary collateral vessels;Altman;Circ Res,1992

2. Determinant of aortic cyclic guanosine monophosphate in hypertension induced by chronic inhibition of nitric oxide synthase;Arnal;J Clin Invest,1992

3. Flow-induced prostacyclin production is mediated by a pertussis toxin-sensitive G protein;Berthiaume;FEBS Letter,1992

4. Pharmacology of a selective cyclooxygenase-2 inhibitor, L-735,337: a novel nonsteroidal antiinflammatory agent (NSAID) with an ulcerogenic sparing effect in rat and nonhuman primate stomach;Chan;J Pharmacol Exp Ther,1995

5. Action of nitric oxide on the release of prostacyclin from bovine endothelial cells in culture;Doni;Eur J Pharmacol,1988

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