Immunoglobulin A nephropathy in a patient with an MYH9 ‐related disorder
Author:
Affiliation:
1. Department of Pediatrics and Child Health Nihon University School of Medicine Tokyo Japan
2. Itabashi Central Medical Center Tokyo Japan
Funder
Japan Agency for Medical Research and Development
Publisher
Wiley
Subject
Pediatrics, Perinatology, and Child Health
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/ped.14628
Reference7 articles.
1. IgA nephropathy and IgA vasculitis with nephritis have a shared feature involving galactose-deficient IgA1-oriented pathogenesis
2. Glomerular IgA deposits in an autopsy study;Suganuma T;Nihon Jinzo Gakkai Shi.,1994
3. Patients with Epstein–Fechtner syndromes owing to MYH9 R702 mutations develop progressive proteinuric renal disease
4. Renin-angiotensin system blockade is effective in reducing proteinuria of patients with progressive nephropathy caused by MYH9 mutations (Fechtner-Epstein syndrome)
5. Polymorphisms in the nonmuscle myosin heavy chain 9 gene (MYH9) are associated with the progression of IgA nephropathy in Chinese
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