Effect of the putative cognitive enhancer, linopirdine (DuP 996), on quantal parameters of acetylcholine release at the frog neuromuscular junction
Author:
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.1994.tb14858.x/fullpdf
Reference42 articles.
1. Physostigmine, but not neostigmine, inhibits acetylcholine release;ALDERDICE;Brain Res.,1979
2. Facilitatory actions of aminopyridines on neuromuscular transmission;BOWMAN;J. Pharm. Pharmacol.,1976
3. Evoked neurotransmitter release: statistical effects of nonuniformity and nonstationarity;BROWN;Proc. Natl. Acad. Sci. U.S.A.,1976
4. Cognition enhancement by the acetylcholine releaser DuP 996;COOK;Drug Dev. Res.,1990
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1. The Contribution of L-Type Calcium Channels to Acetylcholine Secretion in Frog and Mouse Neuromuscular Junctions with Active and Inactivated Voltage-Gated Potassium Channels;Neuroscience and Behavioral Physiology;2020-09
2. Validation of an atomic absorption rubidium ion efflux assay for KCNQ/M-channels using the Ion Channel Reader 8000;ASSAY DRUG DEV TECHN;2004
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4. Sequence dependence of post‐tetanic potentiation after sequential heterosynaptic stimulation in the rat auditory cortex;The Journal of Physiology;2001-06
5. Linopirdine: Pharmacology of a Neurotransmitter Release Enhancer;CNS Drug Reviews;1997-06
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