α2-Adrenergic hyperpolarization is not involved in slow synaptic inhibition in amphibian sympathetic ganglia
Author:
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.1986.tb10831.x/fullpdf
Reference39 articles.
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2. Ionic mechanisms involved in the release of 3H-norepinephrine from the cat superior cervical ganglion;ADLER-GRASCHINSKY;Life Sci,1984
3. Pharmacological properties and monoaminergic mediation of the slow i.p.s.p. in mammalian sympathetic ganglia;ASHE;Brain Res,1982
4. α-adrenergic antagonists as possible calcium channel inhibitors;ATLAS;Proc. natn. Acad. Sci., U.S.A.,1981
5. A functional basis for the classification of α-adrenergic receptors;BERTHELSEN;Life Sci,1977
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1. Amphibian sympathetic ganglia: An owner's and operator's manual;Progress in Neurobiology;1994-07
2. The α1- and α2-adrenoceptor and muscarinic responses of normal and axotomized bullfrog sympathetic ganglia;Canadian Journal of Physiology and Pharmacology;1990-09-01
3. Effects of muscarine and adrenaline on neurones from Rana pipiens sympathetic ganglia.;The Journal of Physiology;1990-06-01
4. Adrenaline inhibits muscarinic transmission in bullfrog sympathetic ganglia;Pflügers Archiv - European Journal of Physiology;1989-04
5. Elevation of intracellular cyclic AMP concentration fails to inhibit adrenaline-induced hyperpolarization in amphibian sympathetic neurones;British Journal of Pharmacology;1989-04
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