Reduction by prostaglandin E1 or prostaglandin E0 of myocardial infarct size in the rabbit by activation of ATP-sensitive potassium channels-Reference-Cited by-同舟云学术

Reduction by prostaglandin E1 or prostaglandin E0 of myocardial infarct size in the rabbit by activation of ATP-sensitive potassium channels

Published:1995-11 Issue:5 Volume:116 Page:2435-2440
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:

Author:

Hide Emma J.,Ney Peter,Piper Julie,Thiemermann Christoph,Vane John R.

Publisher

Wiley

Subject

Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.1995.tb15092.x/fullpdf

Reference48 articles.

1. Role of prostacyclin in the preservation of ischaemic myocardial tissue in the perfused cat heart;ARAKI;Circ. Res.,1980

2. Blockade of ischaemic preconditioning in dogs by the novel ATP dependent potassium channel antagonist sodium 5-hydroxydecanoate;AUCHAMPACH;Cardiovasc. Res.,1992

3. The new K+ channel opener aprikalim (RP 52891) reduces experimental infarct size in dogs in the absence of hemodynamic changes;AUCHAMPACH;J. Pharmacol. Exp. Ther.,1991

4. A critical look at currently used indirect indices of myocardial oxygen consumption;BALLER;Basic Res. Cardiol.,1981

5. Evidence that prostaglandins I2, E2, and D2 may activate ATP-sensitive potassium channels in the isolated rat heart;BOUCHARD;Cardiovasc. Res.,1994

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