Influence of lorcainide on microsomal Na+, K+-ATPase in guinea-pig isolated heart preparations
Author:
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.1991.tb12205.x/fullpdf
Reference26 articles.
1. Antifibrillatory efficacy of encainide, lorcainide and ORG 6001 compared with lignocaine in isolated hearts of rabbits and guinea-pigs;ALMOTREFI;Br. J. Pharmacol.,1981
2. Suggestive evidence for inhibitory action of amiodarone on Na+, K+-pump activity in guinea pig heart;AOMINE;Gen. Pharmacol.,1989
3. Electrophysiological effects of lorcainide, a new antiarrhythmic drug;BÄR;Br. Heart J.,1981
4. Antiarrhythmic treatment: an overview;BIGGER;Am. J. Cardiol.,1984a
5. Perspectives on the current treatment of cardiac arrhythmias;BIGGER;Am. J. Cardiol.,1984b
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1. Inhibition of voltage-dependent K+ channels in rabbit coronary arterial smooth muscle cells by the class Ic antiarrhythmic agent lorcainide;European Journal of Pharmacology;2021-08
2. Class I antiarrhythmic drug effects on ouabain binding to guinea pig cardiac Na+-K+ATPase;Canadian Journal of Physiology and Pharmacology;1999-10-25
3. Class I antiarrhythmic drug effects on ouabain binding to guinea pig cardiac Na+-K+ ATPase;Canadian Journal of Physiology and Pharmacology;1999
4. Actions of amiodarone on mitochondrial ATPase and lactate dehydrogenase activities in guinea pig heart preparations;European Journal of Pharmacology;1993-09
5. Effects of β-adrenoceptor blockers on mitochondrial ATPase activity in guinea pig heart preparations;European Journal of Pharmacology;1992-05
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