Incomplete dissociation of glibenclamide from wild-type and mutant pancreatic KATPchannels limits their recovery from inhibition
Author:
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.2008.00005.x/fullpdf
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4. KATP channels ‘vingt ans apres’: ATG to PDB to Mechanism;Babenko;J Mol Cell Cardiol,2005
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1. The place of gliclazide MR in the evolving type 2 diabetes landscape: A comparison with other sulfonylureas and newer oral antihyperglycemic agents;Diabetes Research and Clinical Practice;2018-09
2. Binding of sulphonylureas to plasma proteins – A KATP channel perspective;PLOS ONE;2018-05-17
3. Importance of the Kir6.2 N-terminus for the interaction of glibenclamide and repaglinide with the pancreatic KATP channel;Naunyn-Schmiedeberg's Archives of Pharmacology;2011-11-15
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