Prejunctional α2-adrenoceptors in mouse atria function through G-proteins which are sensitive to N-ethylmaleimide, but not pertussis toxin
Author:
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.1992.tb14427.x/fullpdf
Reference39 articles.
1. Islet-activating protein (pertussis toxin) diminishes α2-adrenoceptor mediated effects on noradrenaline release;ALLGAIER;Naunyn-Schmiedebergs Arch. Pharmacol,1985
2. Uncoupling of G-aminobutyric acid B receptors from GTP-binding proteins by N-ethylmaleimide: effects of N-ethylmaleimide on purified GTP-binding proteins;ASANO;Mol. Pharmacol,1986
3. Pertussis toxin induced tachycardia and impairs the increase in blood pressure produced by α2-adrenergic agonists;BOYER;Life Sci,1983
4. Pertussis toxin and pre-junctional α2-adrenoceptors in rat heart and vas deferens;DOCHERTY;J. Autonom. Pharmacol,1988
5. No effect of pertussis toxin on peripheral prejunctional α2-adrenoceptor-mediated responses and on endothelium-dependent relaxation responses in the rat;DOCHERTY;Br. J. Pharmacol,1990
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