Pharmacology of a cloned potassium channel from mouse brain (MK-1) expressed in CHO cells: effects of blockers and an ‘nactivation peptide’

Author:

Robertson Brian,Owen David G.

Publisher

Wiley

Subject

Pharmacology

Reference43 articles.

1. Glial- and neuronal-specific K+ channel gene expression in the CNS of the mouse;ADAMS;Proc. Natl. Acad. Sci. U.S.A.,1993

2. Glial- and neuronal-specific K+ channel gene expression in the CNS of the mouse;ADAMS;Soc. Neurosci. Abst.,1991

3. Antiarrythmic properties of tedisamil (KC8857), a putative transient outward K+ current blocker;BEATCH;Br. J. Pharmacol.,1991

4. Simplified gene nomenclature;CHANDY;Nature,1991

5. Tetraethylammonium blockade distinguishes two inactivation mechanisms in voltage-activated K+ channels;CHOI;Proc. Natl. Acad. Sci. USA.,1991

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