Myocardial protection after monophosphoryl lipid A: studies of delayed anti-ischaemic properties in rabbit heart

Author:

Baxter G. F.,Goodwin R.W.,Wright M.J.,Kerac M.,Heads R. J.,Yellon D.M.

Publisher

Wiley

Subject

Pharmacology

Reference44 articles.

1. Adenosine receptor involvement in a delayed phase of myocardial protection 24 hours after ischemic preconditioning;Baxter;Circulation,1994

2. Delayed effects of the endotoxin derivative monophosphoryl lipid A on responses to ischemia-reperfusion in rabbit myocardium;Baxter;Br. J. Pharmacol.,1995

3. Neutrophils contribute to TNF induced myocardial tolerance to ischaemia;Brown;J. Mol. Cell. Cardiol.,1992

4. Endotoxin pretreatment increases myocardial catalase and decreases ischemia-reperfusion injury of isolated rat hearts;Brown;Proc. Natl. Acad. Sci. U.S.A.,1989

5. Endotoxin induces in vivo myocyte hsp70 gene expression and protection from cardiac ischemic injury;Brown;J. Cell. Biochem.,1993

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